تبدلات غارات الدم الشرياني عند مرضي تشمع الكبد والحبن

الموضوعات

طب

روابط وملفات

التعريف والنوع

رقم الوثيقة

02275622-e69e-4972-beb4-71bbea9cb358

رقم العقد

0

نوع الوسائط

Crawler

نوع المحتوى

الرسائل العلمية

صيغة المصدر

رسائل ماجيستير

نوع الملف

pdf text

أسماء الملفات

1963989_1.pdf

بيانات النشر

ترجمة العنوان

Alterations in Arterial Blood Gases in Patients with Liver Cirrhosis and Ascites

ألقاب المؤلفين

[{"name_ar":"أمين شرم ","title_ar":"اعداد","title_en":"Preparation"},{"name_ar":"عبد المسيح مرجانة","title_ar":"اشراف","title_en":"Supervision"}]

اللغة

Arabic

المصدر والدورية

اسم المصدر

تبدلات غارات الدم الشرياني عند مرضي تشمع الكبد والحبن

المحتوى والصفحات

عدد الصفحات

84

ترجمة الملخص

Introduction and Aims:Mechanisms of the development of hypoxemia in cirrhosis are still not well understood. In this study, we aimed to study the Alterations in Arterial Blood Parameters in Patients with Liver Cirrhosis and Ascites, and to investigate and determine the factors contributing to hypoxemia in patients with cirrhosis. .Material and method:From July 2014 to July 2015, We studied 57 cases of liver cirrhosis (LC) compared to 60 normal controls. Causes were: viral 51 (89.4%), alcoholic 1 (1.7%), and cryptogenic 5 (8.7%). Complications were: upper gastrointestinal bleeding 10 (17.5%) "caused by varicose of esophagus, varicose of fundus of stomach" , hepatic encephalopathy 23 (22.8%), Congestive gastropathy 30 (52.6%), HCC 2 (3.5%), hepatorenal syndrome 3 (5.2%) .Samples from artery blood were collected from both patients and controls and all samples were analyzed to measurement the artery blood gases. .Results:Average PaO2 and SaO2 were 78±9 mmHg and 93±7 mmHg, respectively, compared to 95±2 mmHg and 98±1 mmHg of the control group, respectively (P value in both PaO2 and SaO2 was P<0.001). Respiratory alkalosis, metabolic alkalosis, metabolic acidosis, respiratory acidosis and metabolic acidosis with respiratory alkalosis were acid-base disturbances observed.Conclusion: In conclusion, Portopulmonary shunt, intrapulmonary arteriovenous shunt and VA/Q inequality can induce a decrease in PaO2 and SaO2 as well as various acid-base disturbances. As a result, pulmonary resistance is impaired and patients more likely succumb to infections and acute respiratory distress syndrome.

كلمات الباحثين

ascites, Cirrhosis, hepatopulmonary syndrome, hypoxemia

إشراف وإعداد

الإشراف

عبد المسيح مرجانة

الإعداد

أمين شرم

الاقتباسات الببليوغرافية